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Charlie's
Story
By George Glasser and Anita Knight
In 1962, when Anita Knights son, slightly over
a year old, was diagnosed with a lactose intolerance, the doctor prescribed
soy-based (Neo-Mulsoy) infant formula. Within a week of being on the
soy-based infant formula, the baby was vomiting and had severe diarrhea.
Anita wrote, "I changed his bedding and diapers about
five times during the night. The next morning I awoke, shocked to find
him sitting up in the crib with a dead expression on his face. He looked
like a frail little old man because he had lost most of his body fluids.
Terrified, I called the doctor who immediately admitted him to the hospital.
My baby was so dehydrated that they strapped him to an infant-sized
wooden board, put intravenous needles into his arms and legs, and fed
him fluids and nutrients.
"A week later, he was so near death that I was called
to the hospital from work. The pediatrician realized that my son had
suffered a reaction to the soy formula and recommended a diet consisting
of only mashed bananas. After another week, he was well enough to come
home. The diagnosis was gastroenteritis."
In 1989, Anita read an article in the local newspaper
saying that the St. Petersburg City Council had scheduled a meeting
to discuss fluoridation of drinking water. An intriguing statement by
retired dentist Dr. Herbert Robinson, a pioneering fluoride researcher
(and now an opponent of fluoridation), prompted Anita to attend the
meeting. It changed her life and she embarked on a period of research
that continues to this day.
During her studies, Anita came across an article which
was very relevant to her family and her new interest. It said: "Following
an American Dental Association meeting, the manufacturers of infant
formula realized the need to monitor and, in certain cases, reduce the
fluoride ion concentration in their products. . ."1
The article said that fluoride concentrations in soy-based infant formula
were consistently higher than in milk-based products. Realizing that
her son had exhibited classic indicators of severe fluorine poisoning,
Anita worked even harder at her research.
A February 1972 article in Prevention Magazine,
"How Airborne Fluorides Can Poison Foods" made for even more disturbing
reading. It said: "It was in 1968 that the toxic organic fluoride
compoundsfluoroacetate and fluorocitratewere detected
in soy beans that had been fumigated with hydrogen fluoride gas. . ."
There was no monitoring of fluoride levels in the soy infant formula
at the time Anitas son was poisoned. 2, 3,
4, 5, 6, 7, 8
Other studies from 1968 revealed that fluoroacetate
and fluorocitrate were more than 500 times more toxic than inorganic
fluorides. Unfortunately, most prominent researchers and toxicologists
continue to base the toxicity of fluoride in soybeansand of most
species of fluorideson that of sodium fluoride. They do not take
into account any interactions or compound effects. In the case of Anitas
son, the toxic organic fluorides may have triggered a physical chain
reaction that caused the overall effects of the toxicants in the soy-based
infant formula to become potentiated. However, as long as researchers
refuse to consider these possibilities, further progress will never
be made.
Subsequently, researchers testing the organic fluorides
extracted from soy beans discovered that they disrupt carbohydrate metabolism.
Later studies found that fluorocitrate and fluoroacetate produce neurotoxic
effects and kidney, liver and reproductive damage in animals. 9,
10
Fluoroacetate is a natural form of the slightly more
toxic sodium fluoroacetate, also known as the notorious rodent poison
Compound 1080. When ingested and metabolized, fluoroacetate is transformed
in cells to fluorocitratea strong enzyme inhibitor. Fluorocitric
acid blocks the tricarboxylic acid cycle, an essential mechanism of
energy production in mammalian cells.
Compound 1080 is also known as "the poison that keeps
on killing." In other words, the cat that ate the rat that was poisoned
by 1080 has forfeited all its nine lives. For a time, 1080 was banned
in the United States. Today, it can only be used by licensed exterminators,
but environmentalists continue to oppose its use.
For thirty years, Anita believed that she was responsible
for her sons health problems. "When he developed chronic gastroenteritis,
I thought it was all my fault." She recalls that he was ridiculed because
of his fluorosed teeth. "I remember walking into the bathroom and watching
Charlie trying to scrape the stains off his teeth with a sewing needle.
People asked him if he ever brushed his teeth. Now that I know more,
I am even more concerned for Charlies health."
Revelations about the dangers of soy infant formula
were discussed within the scientific community for ten years before
any action was taken by the American Dental Association. Anita wonders
how many children died or still suffer today, like Charlie. "I find
it difficult to believe that prominent researchers remained silent.
They knew that babies were dying and many of those that didnt
die would suffer dental fluorosis and other dreadful, life-long effects
because their mothers gave them soy infant formula."
Today, Anita believes that it was the toxic organic
fluorides that triggered the acute reaction because they are known to
interfere with enzyme functions.
Journals regularly publish new findings about soy infant
formula. It is now known that the product contains high concentrations
of aluminum, an acknowledged neurotoxic substance. Current research
links the aluminum/fluoride combination with neurotoxicity and kidney
damage.
Other problem areas include: phytates, which block the
bodys uptake of minerals; enzyme inhibitors, which hinder protein
digestion; and haemagglutinin, which causes red blood cells to clump
together and inhibits oxygen take-up and growth. Soy also contains high
levels of the phytoestrogens (also known as isoflavones) genistein and
daidzein, which mimic and sometimes block the hormone estrogen.
Charlie, who nearly died in infancy after being fed
soy infant formula for one week, will carry a legacy throughout his
lifedental fluorosis, severe and chronic gastroenteritis and bone
problems.
References
- DG, Pendrys, JW, Stamm, Relationship of Total Fluoride
Intake to Beneficial Effects and Enamel Fluorosis, Journal of Dental
Research, Feb. 1990, Vol 69.
- LH Weinstein, Boyce Thompson Institute, 24:1961.
- J Lovelace, et al, Atmospheric Environment,
2:1968.
- JY Cheng, et al, Environmental Science and Technology,
Vol. 2:1968.
- MH Yu, GW Miller, Environmental Science and Technology,
4: 492, 1970.
- LH Weinstein, et al, Studies on Fluoro-Organic
Compounds in Plants, Environmental Research, Vol 5. 1972.
- DC McCune, LH Weinstein, Metabolic Effects Of Atmospheric
Fluorides On Plants, Environmental Pollution, Vol. 1 1971
- SN Braen, LH Weinstein, Uptake Of Fluoride and Aluminum
By Plants Grown in Contaminated Soils, Water, Air and Soil Pollution,
24:1985
- Clinical Toxicology of Commercial Products,
N.M. Gleason, et al, Eds., (Williams and Wilkins, Blatimor, 1969).
- (Poisoning from Soy sauce) Meyer, J. J. M.; Grobbelaar,
N.; Steyn, P. L. (1990) Fluoroacetate metabolizing pseudomonad isolated
from Dichapetalum cymosum. Applied Environmental Microbiology
56 (7, Jul):2152-2155

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