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You may be low dose of naltrexone. If fifty milligrams blocks phins; second, civilized peoples are addicted to
surprised to the opiate receptors for a day, he reasoned, then substances that stress the adrenal glands, such
three or four milligrams will block the receptors as coffee, tea, chocolate, sugar and stronger
learn that for about an hour. We give the dose at bedtime drugs—you might say that the process of be-
Iscador is the and the body says, “Hey, somebody blocked my coming civilized takes us from the slow lane to
most endorphin sites! I need to make more endor- the fast lane—and as the adrenals are involved
phins.” Sometimes there is a ten-fold increase in endorphin production, with so much stress
prescribed in the number of endorphins produced. The and over-use, our innate feel-good mechanism
cancer next thing you know you find a normal or even breaks down. Finally, civilization puts millions
medicine in heightened response in endorphin production of people into jobs they can’t stand, relationships
leading to improved immune function. In one that are stressful, activities they don’t enjoy.
the world. survey, forty out of forty-two MS patients went Civilization is interesting and challenging, but
into remission using LDN. Their autoimmune it is also stressful.
disease had been based on toxic opiates replacing We often hear of a person diagnosed with
healthy endorphins in their immune response. cancer who says to himself, “Well, if I have only a
There are many classes of diseases that have been few months to live, I’m going to do what I always
helped with this therapy and you can find much wanted to do.” So he quits his work and plays the
more information at www.lowdosenaltrexone. cello, or takes up oil painting. And lo and behold,
org. his cancer goes into remission. Why? Because
How does the use of LDN fit into our theory his body is finally producing and benefitting from
that cancer is a disease of civilization? First, endorphins, his immune system can finally work
the foods of civilization, especially the current again, and he gets well.
lowfat (or wrong-fat) and low-cholesterol diet, It is interesting to compare this therapy to
impede the body’s production of natural endor- the GAPS diet, which eliminates the disaccha-
SOME RECENT STUDIES INVOLVING MISTLETOE EXTRACT
This study showed that complementary treatment with sME [a mistletoe extract] can beneficially reduce the side-effects
of chemotherapy in cancer patients and thus improve quality of life (Anticancer Res 2004 Jan-Feb;24(1):303-9).
The results of this study show that sensitivity to IscadorQu [a mistletoe extract] treatment varies strongly between different
cell lines. In sensitive cell lines, including tumor and endothelial cell cultures, IscadorQu caused early cell cycle inhibition
followed by apoptosis in a dose-dependent manner (Int J Oncol 2004 Dec;25(6):1521-9).
Complementary treatment of breast cancer patients with lectin-standardized mistletoe extract (sME) proved to be a well
tolerated optimization of standard tumor-destructive therapies, mainly improving quality of life and relapse-free intervals
in defined UICC stages (Anticancer Res 2003 Nov-Dec;23(6D):5081-7).
Mistletoe extracts have immunomodulatory activity. We show that nontoxic concentrations of Viscum album [mistletoe]
extracts increase natural killer (NK) cell-mediated killing of tumor cells but spare nontarget cells from NK lysis (Eur J Bio-
chem 2002 May;269(10):2591-600).
Results from the present study suggest that VA [an extract of mistletoe] extract-induced endothelial apoptosis may explain
the tumor regression associated with the therapeutic use of VA preparations and support further investigations to develop
novel anti-angiogenic compounds based on mistletoe compounds (Mol Med 2002 Oct;8(10):600-6).
These results demonstrate the presence of insulin-releasing natural product(s) in Viscum album [mistletoe] which may
contribute to the reported antidiabetic property of the plant (J Endocrinol 1999 Mar;160(3):409-14).
Selective apoptotic effects of VAA-I [a mistletoe extract] may represent a novel approach for pharmacological manipu-
lation of the balance between cell growth and programmed cell death. Appropriate combination of immunomodula-
tory and cytotoxic doses may open new clinical perspectives in the mistletoe therapy (Forsch Komplementarmed 1999
Aug;6(4):186-94).
30 Wise Traditions WINTER 2009